Popular Products Visit the Store. Product Description. Made in USA Consistent Quality Formulation Made in USA Made in Good Manufacturing Practice Facility Consistent Manufacturing Quality Quality natural ingredients No artificial ingredients or preservatives 3rd party lab tested.

High Quality Made in USA Formulation PureMax Labs Green Tea Extract capsules are made in the USA in an FDA inspected, Good Manufacturing Practice Certified GMP facility from premium non-GMO ingredients.

Other premium supplements from PureMax Labs you may like:. Green Tea Extract Olive Leaf Extract Hawthorn Berry Extract Black Seed Oil Blood Circulation Support Thyroid Support Benefits Boost energy, brain function, immune support.

immune support. Compare with similar items This Item. YUMMYVITE Noni Capsules - Concentrated Noni Fruit Extract Superfood Morinda Citrifolia Equivalent to mg Noni Fruit, Immune Support - 60 Capsules.

YUMMYVITE Organic Ceylon Cinnamon - mg per Serving, Certified Organic True Cinnamon Supplement, antioxidant, Fast Absorption - 60 Tablets. Built By Nature. Axio Supply. Green Tea Extract.

Noni Fruit Extract. Organic ceylon cinnamon powder Cinnamomum verum quills. Other Ingredients: Hypromellose cellulose capsule , Microcrystalline Cellulose, Stearic Acid vegetable source and Silicon Dioxide.

Not manufactured with yeast, wheat, gluten, soy, milk, egg, fish, shellfish, tree nut or sesame ingredients. Produced in a GMP facility that processes other ingredients containing these allergens.

Caution: For adults only. Take with food. Keep out of reach of children. Natural color variation may occur in this product. Made and quality tested in the USA with globally sourced ingredients. Store in a cool, dry place after opening.

Family owned since Camellia Sinensis Leaf. Green Tea. Brief content visible, double tap to read full content. Full content visible, double tap to read brief content. Help others learn more about this product by uploading a video! Legal Disclaimer Statements regarding dietary supplements have not been evaluated by the FDA and are not intended to diagnose, treat, cure, or prevent any disease or health condition.

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Easy To Swallow, No Taste Or Smell, A Bump In Energy!! This is a medium sized capsule, which is easy to swallow. I detected no aftertaste, and there was no smell. I'm taking this supplement in the morning due to the caffeine content. It's only a small amount of caffeine, but it IS there - and I'm a little sensitive to caffeine.

That being said, the little boost of energy I get is just right! I recommend this supplement. Will order again.

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These really do help with weight loss. No taste. I love it. I take it every day. Seems to be a decent green tea supplement.

Not really noticing the benefits but it may be different for somebody else. Es fácil de tratar no tiene sabor. One person found this helpful. Translate review to English. Awful still keeping a. Bag next to me last night i thew up took 1 and this morning and got a bag for and night throwing all day I was sick for 3 days hard to swallow.

Vine Customer Review of Free Product What's this? This is a minimally processed green tea extract. Green tea has historically been enjoyed as, well, a tea. With most biologicals in most cases, I think doing too much processing to concentrate this or that compound is interesting in a technical sense but unnecessary.

Not enough to cause any side effects for most, and enough for the proven benefits of EGCG to manifest themselves over time.

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Recommended Uses For Product. Boost energy, brain function, immune support. Heart health, immune support, antioxidant. Heart health, immune health, blood pressure support.

Cold pressed quality Black Seed oil. dummy YUMMYVITE Noni Capsules - Concentrated Noni Fruit Extract Superfood Morinda Citrifolia Equivalent to mg Noni Fruit, Immune Support - 60 Capsules.

dummy YUMMYVITE Organic Ceylon Cinnamon - mg per Serving, Certified Organic True Cinnamon Supplement, antioxidant, Fast Absorption - 60 Tablets. Details Added to Cart Add to Cart. Price Per Unit. Customer Ratings. B The representative outcome of lifespan assay of skn-1 mutants treated with 0.

D The representative outcome of lifespan assay of daf mutants treated with 0. F P -values are as indicated in the graphs. The major antioxidant enzymes in C. elegans include five distinct superoxide dismutases, converting superoxide to hydrogen peroxide, and two catalases, which ensure the subsequent conversion of hydrogen peroxide to water [ 31 ].

EGCG treatments increased SOD activity after 24 h Figure 5A and CTL activity after 7 days Figure 5B. Meanwhile, ECG treatments did not significantly increase SOD activity Figure 5A but increased CTL activity after 24 h and 7 days Figure 5B.

The enhanced activity of SOD and CTL correlates with the subsequent drop of ROS levels after 24 h of EGCG and ECG treatment. Notably, the lifespan-extending effect of EGCG and ECG is dependent on SOD-2 Figure 5C and catalase 2 CTL-2 Figure 5D. As shown in Figure 3 , complex I inhibition by EGCG and ECG was also accompanied by a reduction in glucose oxidation.

In line with this finding, the fat content was found to be significantly lower after h of EGCG or ECG treatment Figure 5E , pointing to a catechin-induced long-term reprogramming of cellular metabolism. Figure 5. EGCG and ECG induce SOD and CTL activity and a shift in lipid metabolism in the long term.

SOD A or CTL B activity after treatment with 0. The representative outcome of lifespan assay of sod-2 mutants treated with 0. C The representative outcome of lifespan assay of ctl-2 mutants treated with 0. D Triglyceride content in N2 wild-type nematodes after treatment with 0. E P -values are as indicated in the graphs.

Green tea is one of the most widely consumed beverages worldwide [ 32 ]. The popularity of green tea makes it crucial to study its impact on health and aging.

Previous reports already reported a lifespan extension in C. elegans after treatment with 50— μM EGCG [ 11 ]. Here, we show that already 2. In this mitohormetic response, EGCG and ECG act initially as prooxidants by provoking a ROS rise. Since a transient ROS burst induces antioxidant defense mechanisms, EGCG and ECG display antioxidant properties in the long term.

In higher concentrations, EGCG and ECG might show harmful effects due to excessive ROS production. This phenomenon gets obvious in studies performed on cancer cells. While the antioxidant potential of green tea catechins in low concentrations was suggested as a potential solution to prevent tumorigenesis [ 34 , 35 ], higher dosages of catechins might serve as antitumor agents due to the induction of overwhelming ROS formation and apoptosis [ 36 — 41 ].

Notably, EGCG was more potent than ECG in human cancer cell lines in inducing cytotoxic effects [ 33 ] and inhibiting cancer cell motility [ 42 ]. Indeed, it took just 6 h for EGCG, but 12 h for ECG to affect mitochondrial respiration, ROS, and ATP levels. However, the impact of these compounds was similar when applied in the long term, yielding similar effects on lifespan, motility, and stress resistance.

Besides triggering a mitohormetic response through their effects on transcription factors and enzyme activities, catechins were speculated to exert direct antioxidant potential by scavenging ROS [ 43 , 44 ].

While a modest increase in the plasma antioxidant capacity following green tea consumption was reported [ 43 ], the fraction of structurally intact catechins reaching target tissues is insignificant compared to the antioxidant potential due to intracellular glutathione achieving levels of 1—11 mM [ 45 — 47 ].

Besides, EGCG even induced hydrogen peroxide formation in the cell culture and liquid NGM system [ 44 — 46 ]. Moreover, hydrogen peroxide mimicked the effect of EGCG on signaling pathways, while antioxidants abolished the impact of catechins [ 37 , 41 , 48 — 50 ]. We could show that BHA prevented lifespan extension by EGCG and ECG, suggesting that an initial rise in ROS levels is necessary to induce adaptational mechanisms causing improved antioxidant properties.

Previous studies already revealed increased hydrogen peroxide levels and a dose- and time-dependent decrease in glutathione levels in cell culture models after applying 50 μM of EGCG [ 43 , 51 ].

However, the mechanism of how EGCG and ECG induce ROS formation was not described so far [ 11 ]. In the current study, we revealed that EGCG and ECG inhibit complex I of the ETC.

This finding is well aligned with the plethora of literature describing polyphenols as compounds targeting mitochondria [ 53 , 54 ]. Consequently, we isolated mitochondria to investigate the impact of EGCG and ECG on the complexes of the mitochondrial ETC.

Isolated mitochondria are separated from their natural environment and signaling processes, and the isolation process brings the risk of damaging mitochondrial membranes due to shear forces [ 55 ].

However, drug uptake by mitochondria is dependent on the integrity of the outer and inner mitochondrial membrane, including the function of transporter proteins and carriers [ 56 ]. Besides, the isolation of mitochondria yields a relatively homogenous population of spherical organelles with disorganized cristae and diluted matrix content.

The structural alterations affect ETC activity and mitochondrial respiration rate [ 57 ]. We assume that structural changes in cristae organization due to the isolation process might be another reason why 25 μM of EGCG and ECG were necessary to significantly block complex I activity and mitochondrial respiration rate in isolated mitochondria.

In addition, we present that a temporary hampered mitochondrial respiration goes along with a transient rise in ROS levels and a brief drop in ATP, triggering signaling pathways associated with lifespan extension in C.

Our findings align with reports about the C. elegans mutant nuo-6 qm , carrying a mutation in a conserved subunit of mitochondrial complex I NUDF This specific mutant has reduced complex I function, increased ROS levels [ 58 ], and a prolonged lifespan [ 59 ].

It was also speculated that blockage of the complex I of the mitochondrial electron transport chain delays aging due to slowed embryonic development and larval growth, decreased pumping and defecation rate, or a reduced accumulation of ROS damage [ 60 — 62 ].

At this stage, mitochondria are already undergoing a period of dramatic proliferation and massive mitochondrial DNA expansion [ 63 ].

Moreover, inhibiting respiratory chain components during adulthood did not provoke lifespan extension anymore [ 64 — 66 ]. Consequently, one has to assume that a temporary sub-lethal rise in mitochondrial ROS during early adulthood induces lifespan extension by provoking changes in the homeostasis of proteins [ 59 , 67 ] and metabolism [ 58 ].

Notably, glucose restriction by 2-deoxy-D-glucose 2-DG -mediated inhibition of glycolysis increases the lifespan in C. elegans in a ROS-dependent manner [ 18 ], suggesting that the temporary drop in ATP levels due to complex I inhibition is an additional trigger to prolong lifespan.

By activating these signaling cascades, the function of ROS defense enzymes, SOD and CTL, and the oxidative stress resistance gets boosted. Ahead of this report, SOD-3, DAF, and SKN-1 were already suggested as targets of EGCG due to enhanced expression [ 68 ] or translocation into the nucleus after respective compound treatment [ 48 ].

Notably, SKN-1 activation in neurons is necessary for dietary restriction-mediated lifespan extension [ 69 ]. DAF, the orthologue of mammalian FOXO, is a crucial regulator of longevity, metabolism, and dauer diapauses in C.

Consequently, it seems reasonable that the ROS-sensing p38 MAPK and the energy-sensing AMPK activate the respective signaling cascades after blockage of complex I by EGCG and ECG. Reports showed that AMPK activates p38 MAPK [ 73 ].

The long-term effects also included reduced fat content in C. elegans after 5 days of catechin treatment. Align with this finding, inhibition of complex I and complex IV by rotenone and NaN3 reduced lipid accumulation in 3T3-L1 cells [ 74 ].

Moreover, a previous report revealed reduced body fat content in C. elegans after catechin treatment [ 75 ]. Besides, green tea catechins were associated with reduced obesity in zebrafish [ 76 ], mice [ 77 ], rats [ 78 , 79 ], and humans [ 80 , 81 ], suggesting a catechin-induced metabolic remodeling.

Clinical trials have already confirmed the safety of EGCG [ 7 ] and highlighted the potential in counteracting age-related cardiovascular and metabolic diseases [ 1 — 4 ]. Experiments in rodents studying physical and clinical parameters over time and further clinical trials are required to identify the best timing and dosage for administering catechins.

Finally, these studies might characterize additional effects and downstream mechanisms of complex I inhibition. Despite the promising results obtained in animal experiments, the low bioavailability of EGCG [ 7 ] still raises the question of whether green tea catechins can reliably provoke beneficial effects in humans.

Consequently, additional efforts might be needed to identify complex I inhibitors with increased bioavailability. We conclude that applying the green tea catechins EGCG and ECG at a low dose extends the lifespan of C. elegans via inducing a mitohormetic response. In the long term, the re-wiring of these energy- and ROS-dependent pathways reduces the fat content and extends health- and lifespan.

Figure 6. Green tea catechins enhance fitness and lifespan of Caenorhabditis elegance by complex I inhibition. elegans strains used in the current study were obtained from the Caenorhabditis Genetics Center CGC, University of Minnesota.

Nematodes were grown and maintained at 20°C in 10 cm Petri dishes on nematode growth media NGM , with Escherichia coli E. coli OP50 bacteria as the food resource as previously described [ 18 , 82 , 83 ]. The strains used in this study included the following: N2 wild type , GA aak-2 ok , VC sir EGCG, ECG, and BHA dissolved in DMSO, reaching a stock concentration of 2.

The NGM agar solution was autoclaved and subsequently cooled to 55°C, before supplements and compounds EGCG, ECG, BHA, or DMSO were added under continuous stirring. The final concentration for compounds was calculated regarding the volume of agar, and the same volume of DMSO was added to control plates.

Agar plates were poured and dried, sealed with parafilm, and stored at 4°C. Before experiments, NGM plates were spotted with a bacterial lawn of heat-inactivated bacteria OP50 HIT to avoid interference by a potential xenobiotic-metabolizing activity of E. To exclude any effects on development, the incubation period with compounds started at the L4 stage by transferring nematodes to the respective NGM plates [ 84 ].

Louis, MO, USA to prevent progeny formation. After 16 h, we transferred animals to respective treatment groups and harvested them at the indicated time points [ 18 ]. According to standard protocols, all lifespan assays were performed at 20°C as previously described [ 18 , 19 ].

Briefly, the C. Eggs of nematodes were transferred to NGM plates with fresh OP50 bacteria to allow hatching and development. After approximately 64 h, at the L4 stage, we moved nematodes manually to freshly prepared NGM plates containing the respective compounds and supplied them with a lawn of OP50 HIT.

During the first 10—14 days, nematodes were transferred to freshly prepared NGM treatment plates every day and later every second day. Nematodes without any reaction to gentle stimulation were classified as dead.

Nematodes that crawled off the plate or suffered from non-natural death like internal hatching were censored and excluded from statistics on the day of premature death.

Notably, for lifespan analysis using BHA, nematodes were propagated on BHA-containing NGM plates for four generations before synchronization; the same applied for the respective DMSO controls. Following the L4 stage, nematodes were treated with 0. Afterward, we transferred single worms into S-buffer containing 0.

Movements of single worms within the liquid system were recorded for 20 seconds by a digital CCD camera Moticam , Motic, St. Ingbert, Germany coupled microscope SMZ , Motic, St. Ingbert, Germany equipped with Motic Images Plus 2. We analyzed the videos using the DanioTrack software Loligo Systems, Tjele, Denmark , subtracting the background and determining the center of gravity of all object pixels compared to the background.

Resistance to lethal oxidative stress by paraquat Sigma-Aldrich, Munich, Germany was assessed as previously described [ 18 , 19 ]. Briefly, worms were treated with 0.

Afterward, we transferred worms into well plates: 6 nematodes in μl of S-buffer, containing freshly dissolved 50 mM paraquat. Dead worms were scored every hour until all control worms were dead.

Briefly, we treated worms with 0. Worms were also washed twice with S-buffer and transferred into the DW1 chamber to monitor oxygen consumption for 10 mins. Afterward, we collected worms for Bradford protein determination [ 86 ]. Before the ROS measurement, MitoTracker Red CM-H2X ROS Invitrogen, Carlsbad, CA, USA incubation plates were prepared as previously described [ 19 ].

Worms were additionally washed twice with S-buffer and transferred to freshly prepared MitoTracker Red CM-H2X incubation NGM plates containing μl of OP50 HIT mixed with μl freshly prepared MitoTracker Red CM-H2X stock solution μM.

After 2 h at 20°C, worms were washed off MitoTracker Red CM-H2X incubation NGM plates and transferred to NGM agar plates with 0. Fluorescence intensity was measured on a microplate reader FLUOstar Optima, BMG Labtech, Offenburg, Germany using well-scanning mode ex: nm; em: nm. We collected worms from plates for Bradford protein determination [ 86 ].

We placed an equal number of nematodes on the NGM plates containing 0. After collection and two subsequent washes in S-buffer, worm pellets were resuspended in the incubation buffer. The latter were placed in 10 cm Petri dishes together with a second 4 cm Petri dish containing μl of 0.

Hence, each 10 cm dish was equipped with two 4 cm dishes, one carrying nematodes and the other containing KOH. We added nonradioactive glucose into each sample to reach a final concentration of 0. The 10 cm Petri dishes were covered, sealed with parafilm in an air-tight manner, and incubated at 20°C for 3 h.

Subsequently, an aliquot of μl of KOH was immersed in 4. to quantify the amount of trapped 14 CO 2. We treated nematodes with 0. After collection and washing with S-buffer twice, worm pellets were shock frozen in liquid nitrogen and grinded in a nitrogen-chilled mortar.

The grinded samples were boiled with 4 M Guanidine-HCl at 99°C for 15 min to destroy ATPase activity [ 58 , 89 ]. ATP values were normalized to protein content using the Bradford assay [ 86 ].

After treating nematodes with 0. The produced formaldehyde was determined spectrophotometrically with 4-aminohydrazinomercapto-1, 2, 4-triazole Purpald, Applichem, Darmstadt, Germany. We measured SOD activity photometrically with a tetrazolium salt, forming a water-soluble formazan dye upon reduction with a superoxide anion.

We determined fat content by applying a triglyceride determination kit Roche, Mannheim, Germany as previously described [ 18 , 88 ] and normalized to protein content using the Bradford assay [ 86 ]. Briefly, worms were incubated with 0.

We centrifuged μl of the homogenized extract and extracted the supernatant for protein determination. The heating was repeated once to dissolve all triglycerides. We measured the activity of complex I spectrophotometrically at nm in 1 ml of 25 mM potassium phosphate buffer containing 3. Decylubiquinone and antimycin A were dissolved in DMSO as DCIP and NADH were dissolved in water as 10 mM for both.

After being thawed, 30 μl of mitochondria were treated with μl of 10 mM Tris-Cl, pH 7. Subsequently, 20 μl mitochondria fragments were preincubated in a μl incubation mixture without NADH for 3 mins.

After 3 mins, we added 20 μl of 10 mM NADH into the incubation mixture and measured the absorbance at 20 s intervals for 2 mins. Briefly, rodents were fasted overnight and killed by cervical dislocation.

The washed liver fragments were placed into the tube with around 25 ml isolation buffer. The loose-fitting pestle was inserted, pressed down, and lifted four times, and then the tight-fitting pestle was applied in the same way twice.

The mixture was poured into the 50 ml polypropylene falcon tube and centrifuged at g for 10 min at 4°C. We carefully removed the fat on the top of the supernatant by using tissue paper. The supernatant was extracted to a second polypropylene falcon tube centrifuged at g for 10 min at 4°C.

Afterward, the fat was removed, the supernatant discarded, and the mitochondrial pellet resuspended in the remaining buffer. The suspension containing mitochondria was centrifuged again at g for 10 min at 4°C.

The supernatant was removed entirely, and the mitochondrial pellet was resuspended in μl isolation buffer as described above. The concentration of isolated mitochondria was determined with Bradford After recording basal respiration for 2 min, 0.

After ADP was wholly consumed, the oxygen consumption rate slowed down, 5 mM succinate, and ADP were added to study complex II, III, IV activity. At the end of each measurement 60 nM FCCP were supplied to check the viability of mitochondria. Data are expressed as means ±SD unless otherwise indicated.

It contains antioxidant polyphenols like chlorogenic acid, plus as much caffeine as coffee or more 80 to mg per cup. Preliminary research suggests it might promote weight loss and lower blood cholesterol, but studies are inconclusive.

Users report less fatigue and better focus — likely from its caffeine content — but without jitteriness. Downside: Certain processing methods of mate, such as drying the leaves with smoke, may introduce polycyclic aromatic hydrocarbons — the same carcinogenic substances that are found in grilled meats.

Some research links drinking large amounts of mate over time with increased risk of certain cancers , including head and neck, stomach, bladder, and lung.

However, unsmoked mate which is processed by air drying may be safer. Like mate, yaupon is an herbal tea. Native to the US, it has a mellow grassy flavor similar to green tea. It contains chlorogenic acid and antioxidants that are purported to decrease inflammation and boost energy.

This tea has 60 mg caffeine per cup and also provides theobromine, a compound structurally similar to caffeine found in cocoa beans and many teas. Theobromine increases blood flow and may increase energy and alertness, but this boost is slower to start and lasts longer than caffeine, which provides a quick but short-lived boost.

Downside: The combination of theobromine and caffeine may increase heart rate and interfere with sleep, especially if you drink a large amount of yaupon or sip it too close to bedtime. Matcha comes from the same Camellia sinensis plant as green tea.

However, unlike green tea, matcha is grown in the shade, which protects it from sunlight and oxidation and contributes to its bright green color and higher polyphenol content.

Whole tea leaves and stems of matcha are ground into a fine powder, which is then whisked with hot water or milk. Product Description. Made in USA Consistent Quality Formulation Made in USA Made in Good Manufacturing Practice Facility Consistent Manufacturing Quality Quality natural ingredients No artificial ingredients or preservatives 3rd party lab tested.

High Quality Made in USA Formulation PureMax Labs Green Tea Extract capsules are made in the USA in an FDA inspected, Good Manufacturing Practice Certified GMP facility from premium non-GMO ingredients. Other premium supplements from PureMax Labs you may like:. Green Tea Extract Olive Leaf Extract Hawthorn Berry Extract Black Seed Oil Blood Circulation Support Thyroid Support Benefits Boost energy, brain function, immune support.

immune support. Compare with similar items This Item. YUMMYVITE Noni Capsules - Concentrated Noni Fruit Extract Superfood Morinda Citrifolia Equivalent to mg Noni Fruit, Immune Support - 60 Capsules. YUMMYVITE Organic Ceylon Cinnamon - mg per Serving, Certified Organic True Cinnamon Supplement, antioxidant, Fast Absorption - 60 Tablets.

Built By Nature. Axio Supply. Green Tea Extract. Noni Fruit Extract. Organic ceylon cinnamon powder Cinnamomum verum quills. Other Ingredients: Hypromellose cellulose capsule , Microcrystalline Cellulose, Stearic Acid vegetable source and Silicon Dioxide.

Not manufactured with yeast, wheat, gluten, soy, milk, egg, fish, shellfish, tree nut or sesame ingredients. Produced in a GMP facility that processes other ingredients containing these allergens. Caution: For adults only.

Take with food. Keep out of reach of children. Natural color variation may occur in this product. Made and quality tested in the USA with globally sourced ingredients. Store in a cool, dry place after opening.

Family owned since Camellia Sinensis Leaf. Green Tea. Brief content visible, double tap to read full content. Full content visible, double tap to read brief content.

Help others learn more about this product by uploading a video! Legal Disclaimer Statements regarding dietary supplements have not been evaluated by the FDA and are not intended to diagnose, treat, cure, or prevent any disease or health condition.

Looking for specific info? Customer reviews. How customer reviews and ratings work Customer Reviews, including Product Star Ratings help customers to learn more about the product and decide whether it is the right product for them. Learn more how customers reviews work on Amazon. Images in this review.

Reviews with images. See all photos. All photos. Easy To Swallow, No Taste Or Smell, A Bump In Energy!! This is a medium sized capsule, which is easy to swallow. I detected no aftertaste, and there was no smell. I'm taking this supplement in the morning due to the caffeine content.

It's only a small amount of caffeine, but it IS there - and I'm a little sensitive to caffeine. That being said, the little boost of energy I get is just right! I recommend this supplement. Will order again.

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Top reviews from the United States. Translate all reviews to English. There was a problem filtering reviews right now. Please try again later. Verified Purchase. These really do help with weight loss. No taste. I love it. I take it every day.

Cardiovascular disease: Consumption of red wine has been associated with a reduction in endothelin-1 a molecule involved in blood pressure regulation , a reduction in myocardial ischemic reperfusion injury an injury to the heart when blood is returned to the organ after a period of restriction , increased HDL concentrations, decreased platelet aggregation clumping , increased fibrinolysis breakdown of a clot , and increased plasma antioxidant activity 4,5.

Furthermore, results from some studies indicate that consumption of red wine may slow the progression of atherosclerosis. Diabetes Mellitus: The flavanols in red wine may improve the lipid profile in some individuals. Insulin sensitivity and reduced insulin resistance has been reported to improve in individuals with moderate wine consumption 5.

In animal studies, increased HDL lipoproteins, lowered levels of ox-LDL, decreased platelet aggregation and improvements in endothelial function have been reported following moderate red wine consumption 6. In a randomized study conducted on individuals with controlled Type II Diabetes, the catechins in the red wine were reported t significantly increase plasma HDL levels by 2.

Lung Cancer: Research studies have focused on the correlation of COPD Chronic Obstructive Pulmonary Disease and increased lung cancer risk. Consistent with its putative antioxidant abilities, moderate consumption of red wine has been associated with a reduced risk of lung cancer in comparison to individuals who do not consume red wine 8.

Prostate Cancer: There have been contradicting results regarding consumption of red wine and cancer. Results from some studies suggest that consumption of red wine over a lifetime posed increased risks of prostate cancer.

Further research is needed to better understand the amount and time period of red wine consumption and the associated risks to prostate cancer 9. Conclusions: In light of this research, the American Heart Association does not recommend consumption of alcohol to reduce risk of cardiovascular disease and the American Cancer Society recommends limiting consumption of alcoholic beverages.

If adults choose to drink alcoholic beverages, the Dietary Guidelines for Americans, recommends they do so in moderation.